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For example, only one molecule of cholesterol can pass through the hydrophobic tunnel at a time, which seems inconsistent with the acute steroidogenic response to stimulationof the cholesterol transporter, but this is not really an unexpected finding. Instead, the authors say, these results suggest that one type of steroid is needed (the «compound of interest») and that it must be «selective» in order to trigger this response.

So in conclusion, the authors say, «the evidence for a steroidogenic mechanism within the liver that is mediated by a steroid-metabolizing enzyme is strong.» In that vein, they say that further study is needed to rule out various other compounds as active steroids, such as other drugs that may have similar effects. If the «compound of interest» were actually a steroids metabolite, it would explain their specific actions. But the authors say they know of no such thing—and it would be interesting to know whether this substance is unique to the liver as an organ but not in other animals’ bodies.

[via Nature Cell Biology]

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